Vitamin B12 Deficiency

Definition | Aetiology | Pathophysiology | Risk Factors | Signs and Symptoms | Investigations | Management

Definition

Vitamin B12 deficiency is a condition resulting from inadequate levels of vitamin B12, leading to impaired DNA synthesis, haematological abnormalities, and neurological dysfunction.

Aetiology

  • Pernicious anaemia: autoimmune destruction of gastric parietal cells leading to intrinsic factor deficiency.
  • Dietary deficiency: common in vegans and those with poor nutritional intake.
  • Malabsorption: gastric bypass surgery, coeliac disease, Crohn’s disease.
  • Drug-induced: metformin, proton pump inhibitors (PPIs), H2-receptor antagonists.
  • Increased demand: pregnancy, chronic haemolysis.

Pathophysiology

  • Vitamin B12 is required for DNA synthesis and myelin formation.
  • Deficiency leads to ineffective erythropoiesis, causing megaloblastic anaemia.
  • Neurological complications arise due to impaired methylation of myelin proteins.

Risk Factors

  • Strict vegan diet.
  • History of gastrointestinal surgery or disorders.
  • Long-term use of metformin or PPIs.
  • Age over 65 years.
  • Autoimmune conditions (e.g., type 1 diabetes, thyroid disease).

Signs and Symptoms

  • Fatigue, pallor.
  • Glossitis, angular cheilitis.
  • Neurological symptoms: paraesthesia, numbness, peripheral neuropathy.
  • Neuropsychiatric changes: memory impairment, depression.
  • Gait disturbance, ataxia.

Investigations

  • Full blood count (FBC): macrocytic anaemia.
  • Blood film: hypersegmented neutrophils.
  • Serum vitamin B12: low levels confirm deficiency.
  • Intrinsic factor antibodies: positive in pernicious anaemia.
  • Methylmalonic acid and homocysteine: elevated in B12 deficiency.
  • Coeliac screen: if malabsorption is suspected.

Management

Vitamin B12 Management Summary

  • Indication for Checking B12: Anaemia or strong clinical suspicion (e.g., visual disturbance, memory loss, psychiatric abnormalities, peripheral nerve dysfunction without other identifiable cause).

B12 Level Interpretation:

  • ≥180 ng/L & No Symptoms: No further investigation.
  • 145-179 ng/L:
    • If asymptomatic → Continue dietary control, no further action or they can purchase Oral cyanocobalamin 50-150mcg daily.
    • If strong clinical suspicion persists → Check Intrinsic Factor Antibody (IFAB).
  • <145 ng/L: Check IFAB but proceed to treatment regardless.

IFAB Testing Outcomes:

  • Positive: Confirmed pernicious anaemia.
  • Negative: Consider dietary deficiency.

Management Based on Symptoms:

  • No Neurological Symptoms:
    • Initial: IM Hydroxocobalamin 1mg three times a week for 2 weeks.
    • Maintenance:
      • Dietary cause: Oral cyanocobalamin 50-150mcg daily (OTC).
      • Non-dietary: IM Hydroxocobalamin 1000mcg every 3 months for life.
  • Neurological Symptoms:
    • Seek Specialist Advice (including IFAB result).
    • Immediate treatment while awaiting advice: IM Hydroxocobalamin 1mg every other day until no symptoms.
    • Maintenance: IM Hydroxocobalamin 1000mcg every 2-3 months for life (or as per specialist recommendation).

Specialist Advice Guidance:

  • Consult appropriate specialist (Gastro, Neurology, Elderly Care).
  • Remote advice usually sufficient.
  • Use NHS advice & guidance platforms if available.
  • Consider haematology referral if Hb does not improve.

1. Vitamin B12 Replacement:

  • Hydroxocobalamin 1 mg IM injections: loading dose (3 times a week for 2 weeks), then maintenance every 3 months.
  • Oral cyanocobalamin: for dietary deficiency in non-neurological cases.

2. Treat Underlying Cause:

  • Manage malabsorptive conditions (e.g., treat coeliac disease, adjust medications affecting absorption).
  • Lifelong supplementation in pernicious anaemia.

3. Referral:

  • Haematology: for severe cases or unclear aetiology.
  • Gastroenterology: if malabsorption or coeliac disease is suspected.
  • Neurology: if neurological deficits persist despite treatment.